Serum Complement Activation of SLE Patients During Plasmapheresis
Author:
Publisher
Informa UK Limited
Subject
Immunology,Immunology and Allergy
Link
http://www.tandfonline.com/doi/pdf/10.3109/08916939709008020
Reference19 articles.
1. Inherited deficiency of erythrocyte complement receptor type 1 does not cause susceptibility to systemic lupus erythematosus
2. Peripheral catabolism of CR1 (the C3b receptor, CD35) on erythrocytes from healthy individuals and patients with systemic lupus erythematosus (SLE)
3. The Role of Complement and Its Receptor in the Elimination of Immune Complexes
4. The erythrocyte–immune complex–glomerulonephritis connection in man
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1. Immune complex clearance by complement receptor type 1 in SLE;Autoimmunity Reviews;2008-12
2. The severity of systemic lupus erythematosus negatively correlates with the increasing number of CD4+CD25highFoxP3+regulatory T cells during repeated plasmapheresis treatments of patients;Autoimmunity;2007-01
3. TREATMENT OF C4D-POSITIVE ACUTE HUMORAL REJECTION WITH PLASMAPHERESIS AND RABBIT POLYCLONAL ANTITHYMOCYTE GLOBULIN;Transplantation;2004-05
4. Plasmapheresis Modulates TH1/TH2 Imbalance in Patients with Systemic Lupus Erythematosus According to Measurement of Intracytoplasmic Cytokines;Autoimmunity;2002-01
5. ELISA for evaluating the incorporation of plasma derived complement split-products C3b/iC3b into solid-phase immune complexes;Journal of Immunological Methods;2001-03
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