Lack of Female Bias in a Mouse Model of Autoimmune Hyperthyroidisim (Graves' Disease)
Author:
Publisher
Informa UK Limited
Subject
Immunology,Immunology and Allergy
Link
http://www.tandfonline.com/doi/pdf/10.3109/08916939908994746
Reference13 articles.
1. Induction of Graves-like disease in mice by immunization with fibroblasts transfected with the thyrotropin receptor and a class II molecule.
2. "Exon-shuffling" maps control of antibody- and T-cell-recognition sites to the NH2-terminal domain of the class II major histocompatibility polypeptide A beta.
3. Molecular cloning, sequence and functional expression of the cDNA for the human thyrotropin receptor
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1. A transgenic mouse that spontaneously develops pathogenic TSH receptor antibodies will facilitate study of antigen-specific immunotherapy for human Graves’ disease;Endocrine;2019-09-27
2. Graves’ orbitopathy occurs sex-independently in an autoimmune hyperthyroid mouse model;Scientific Reports;2018-08-30
3. Thyroid Hemiagenesis in a Thyroiditis Prone Mouse Strain;European Thyroid Journal;2018
4. Toward Better Models of Hyperthyroid Graves' Disease;Endocrinology and Metabolism Clinics of North America;2009-06
5. New insights into antibody-mediated hyperthyroidism;Expert Review of Endocrinology & Metabolism;2006-01
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