Inherent β-Cell Dysfunction Induced by Transgenic Expression of Allogeneic Major Histocompatibility Complex Class I Antigen in Islet Cells
Author:
Publisher
Informa UK Limited
Subject
Immunology,Immunology and Allergy
Link
http://www.tandfonline.com/doi/pdf/10.3109/08916939108997123
Reference41 articles.
1. Diabetes in transgenic mice resulting from over-expression of class I histocompatibility molecules in pancreatic β cells
2. Insulin-dependent diabetes mellitus induced in transgenic mice by ectopic expression of class II MHC and interferon-gamma
3. Diabetes and tolerance in transgenic mice expressing class II MHC molecules in pancreatic beta cells
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1. The tumour-suppressor p53 is not required for pancreatic β cell death during diabetes and upon irradiation;The Journal of Physiology;2008-01-14
2. Widespread expression of an autoantigen-GAD65 transgene does not tolerize non-obese diabetic mice and can exacerbate disease;Proceedings of the National Academy of Sciences;1998-08-18
3. ANIMAL MODELS OF HUMAN DISEASE. Transgenic and Knockout Models of Autoimmunity: Building a Better Disease?;Clinical Immunology and Immunopathology;1996-05
4. The estrogen connection: The etiological relationship between diabetes, cancer, rheumatoid arthritis and psychiatric disorders;Medical Hypotheses;1995-08
5. Inflammation but not autoimmunity occurs in transgenic mice expressing constitutive levels of interleukin-2 in islet β cells;European Journal of Immunology;1992-05
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