Dioxin toxicity, aryl hydrocarbon receptor signaling, and apoptosis—Persistent pollutants affect programmed cell death
Author:
Publisher
Informa UK Limited
Subject
Toxicology
Link
http://www.tandfonline.com/doi/pdf/10.3109/10408444.2010.524635
Reference233 articles.
1. Protein kinase C? activity is involved in the 2,3,7,8-tetrachlorodibenzo-p-dioxin-induced signal transduction pathway leading to apoptosis in L-MAT, a human lymphoblastic T-cell line
2. Non-dioxin-like-PCBs phosphorylate Mdm2 at Ser166 and attenuate the p53 response in HepG2 cells
3. Protein Kinase C (PKC ): A Key Enzyme in T Cell Life and Death
4. 2,3,7,8-Tetrachlorodibenzo-p-Dioxin Counteracts the p53 Response to a Genotoxicant by Upregulating Expression of the Metastasis Marker AGR2 in the Hepatocarcinoma Cell Line HepG2
5. Targeting the extrinsic apoptosis pathway in cancer
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