Protection of Tanshinone IIA to Human Peritoneal Mesothelial Cells (HPMC) through Delaying Cellular Senescence Induced by High Glucose
Author:
Publisher
Informa UK Limited
Subject
Nephrology,Critical Care and Intensive Care Medicine,General Medicine
Link
http://www.tandfonline.com/doi/pdf/10.3109/0886022X.2011.623497
Reference27 articles.
1. Prolonged Exposure to Glucose Degradation Products Impairs Viability and Function of Human Peritoneal Mesothelial Cells
2. Peritoneal Fibrosing Syndrome: Pathogenetic Mechanism and Current Therapeutic Strategies
3. Epidermal growth factor modifies the expression and function of extracellular matrix adhesion receptors expressed by peritoneal mesothelial cells from patients on CAPD
4. High Glucose Induces a Hypertrophic, Senescent Mesothelial Cell Phenotype after Long in vivo Exposure
5. High glucose accelerates the life cycle of the in vivo exposed mesothelium
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1. A comprehensive review of tanshinone IIA and its derivatives in fibrosis treatment;Biomedicine & Pharmacotherapy;2021-05
2. CXCR4-Receptor-Targeted Liposomes for the Treatment of Peritoneal Fibrosis;Molecular Pharmaceutics;2019-05-09
3. Upregulation of MiR-126 Delays the Senescence of Human Glomerular Mesangial Cells Induced by High Glucose via Telomere-p53-p21-Rb Signaling Pathway;Current Medical Science;2018-10
4. Sulfotanshinone IIA Sodium Ameliorates Glucose Peritoneal Dialysis Solution-Induced Human Peritoneal Mesothelial Cell Injury via Suppression of ASK1-P38-mediated Oxidative Stress;Cellular Physiology and Biochemistry;2018
5. Cellular senescence and the senescent secretory phenotype in age-related chronic diseases;Current Opinion in Clinical Nutrition and Metabolic Care;2014-07
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