Therapeutic effect of CNP on renal osteodystrophy by antagonizing the FGF-23/MAPK pathway
Author:
Publisher
Informa UK Limited
Subject
Cell Biology,Molecular Biology,Biochemistry
Link
http://www.tandfonline.com/doi/pdf/10.3109/10799893.2015.1075041
Reference83 articles.
1. Early Skeletal and Biochemical Alterations in Pediatric Chronic Kidney Disease
2. Epidemiological study of orthopedic injuries in hemodialysis patients in Taiwan: a fixed cohort survey, 2004–2008
3. Value of the New Bone Classification System in Pediatric Renal Osteodystrophy
4. Differences in Bone Quality in Low- and High-Turnover Renal Osteodystrophy
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1. The downstream RAF-1 signaling of fibroblast growth factor-23 participates in the osteogenetic effect caused by C-type natriuretic peptide in vitro;Advances in Medical Sciences;2021-03
2. C-type natriuretic peptide stimulates osteoblastic proliferation and collagen-X expression but suppresses fibroblast growth factor-23 expression in vitro;Pediatric Rheumatology;2020-06-09
3. C-type natriuretic peptide attenuates renal osteodystrophy through inhibition of FGF-23/MAPK signaling;Experimental & Molecular Medicine;2019-07
4. C-Type Natriuretic Peptide Dampens Fibroblast Growth Factor-23 Expression Through MAPK Signaling Pathway in Human Mesangial Cells;Journal of Interferon & Cytokine Research;2018-11
5. Fibroblast growth factor-23 may serve as a novel biomarker for renal osteodystrophy progression;International Journal of Molecular Medicine;2018-10-15
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