Prolonged swimming promotes cellular oxidative stress and p66Shc phosphorylation, but does not induce oxidative stress in mitochondria in the rat heart-Reference-Cited by-同舟云学术

Prolonged swimming promotes cellular oxidative stress and p66Shc phosphorylation, but does not induce oxidative stress in mitochondria in the rat heart

Published:2014-10-21 Issue:1 Volume:49 Page:7-16
ISSN:1071-5762
Container-title:Free Radical Research
language:en
Short-container-title:Free Radical Research

Author:

Ziolkowski W.,Flis D. J.,Halon M.,Vadhana D. M. S.,Olek R. A.,Carloni M.,Antosiewicz J.,Kaczor J. J.,Gabbianelli R.

Publisher

Informa UK Limited

Subject

General Medicine,Biochemistry

Link

http://www.tandfonline.com/doi/pdf/10.3109/10715762.2014.968147

Reference67 articles.

1. Regular treadmill exercise restores cardioprotective signaling pathways in obese mice independently from improvement in associated co-morbidities

2. Short Term Training Attenuates Opening of the Mitochondrial Permeability Transition Pore Without Affecting Myocardial Function Following Ischemia-Reperfusion

3. Exercise-induced improvements in myocardial antioxidant capacity: the antioxidant players and cardioprotection

4. Exercise Protects Cardiac Mitochondria against Ischemia–Reperfusion Injury

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