ERK Phosphorylation Is Not Increased in Papillary Thyroid Carcinomas with BRAFV600EMutation Compared to That of Corresponding Normal Thyroid Tissues-Reference-Cited by-同舟云学术

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ERK Phosphorylation Is Not Increased in Papillary Thyroid Carcinomas with BRAFV600EMutation Compared to That of Corresponding Normal Thyroid Tissues

Published:2013-04 Issue:2 Volume:38 Page:89-97
ISSN:0743-5800
Container-title:Endocrine Research
language:en
Short-container-title:Endocrine Research

Author:

Kim Sun Wook,Kim Hee Kyung,Lee Ji In,Jang Hye Won,Choe Jun-Ho,Kim Jung-Han,Kim Jee Soo,Hur Kyu Yeon,Kim Jae Hyeon,Chung Jae Hoon

Publisher

Informa UK Limited

Subject

Endocrinology,General Medicine

Link

http://www.tandfonline.com/doi/pdf/10.3109/07435800.2012.723292

Reference33 articles.

1. Thyroid Cancer Incidence and Survival in the National Cancer Institute Surveillance, Epidemiology, and End Results Race/Ethnicity Groups

2. Increasing incidence of differentiated thyroid cancer in the United States, 1988-2005

3. BRAF Mutation in Papillary Thyroid Cancer: Pathogenic Role, Molecular Bases, and Clinical Implications

4. Mechanism of Activation of the RAF-ERK Signaling Pathway by Oncogenic Mutations of B-RAF

5. BRAF as a potential therapeutic target in melanoma and other malignancies

Cited by 5 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Polypyrimidine Tract-Binding Protein 1 (PTBP1) regulates CD4 T cell Activation independent of its role in proliferation;2022-03-21

2. Retracted : MiR‐141‐3p Suppresses Tumor Growth and Metastasis in Papillary Thyroid Cancer via Targeting Yin Yang 1;The Anatomical Record;2018-11-09

3. Novel clinicopathological and molecular characterization of metanephric adenoma: a study of 28 cases;Diagnostic Pathology;2018-08-16

4. Mps1 is associated with the BRAFV600E mutation but does not rely on the classic RAS/RAF/MEK/ERK signaling pathway in thyroid carcinoma;Oncology Letters;2018-04-24

5. TSH Signaling Overcomes B-RafV600E–Induced Senescence in Papillary Thyroid Carcinogenesis through Regulation of DUSP6;Neoplasia;2014-12

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