Methotrexate reduces HbA1c concentration but does not produce chronic accumulation of ZMP in patients with rheumatoid or psoriatic arthritis-Reference-Cited by-同舟云学术

Methotrexate reduces HbA1c concentration but does not produce chronic accumulation of ZMP in patients with rheumatoid or psoriatic arthritis

Published:2016-01-04 Issue:5 Volume:45 Page:347-355
ISSN:0300-9742
Container-title:Scandinavian Journal of Rheumatology
language:en
Short-container-title:Scandinavian Journal of Rheumatology

Author:

Perdan-Pirkmajer K¹,Pirkmajer S²,Thevis M³,Thomas A³,Praprotnik S¹,Hočevar A¹,Rotar Ž¹,Gašperšič N¹,Sodin-Šemrl S¹,Žibert J⁴,Omersel J⁵,Chibalin AV⁶,Tomšič M¹,Ambrožič A¹

Affiliation:

1. Department of Rheumatology, University Medical Centre Ljubljana, Slovenia

2. Institute of Pathophysiology, Faculty of Medicine, University of Ljubljana, Slovenia

3. Institute of Biochemistry, Centre for Preventive Doping Research, German Sport University Cologne, Germany

4. Faculty of Health Sciences, University of Ljubljana, Slovenia

5. Faculty of Pharmacy, University of Ljubljana, Slovenia

6. Department of Molecular Medicine and Surgery, Integrative Physiology, Karolinska Institutet, Stockholm, Sweden

Publisher

Informa UK Limited

Subject

Immunology,General Medicine,Immunology and Allergy,Rheumatology

Link

https://www.tandfonline.com/doi/pdf/10.3109/03009742.2015.1105290

Reference26 articles.

1. Methotrexate therapy associates with reduced prevalence of the metabolic syndrome in rheumatoid arthritis patients over the age of 60- more than just an anti-inflammatory effect? A cross sectional study

2. Effect of Methotrexate Use and Erythrocyte Methotrexate Polyglutamate on Glycosylated Hemoglobin in Rheumatoid Arthritis

3. Association Between Disease-Modifying Antirheumatic Drugs and Diabetes Risk in Patients With Rheumatoid Arthritis and Psoriasis

4. The antiinflammatory mechanism of methotrexate. Increased adenosine release at inflamed sites diminishes leukocyte accumulation in an in vivo model of inflammation.

5. 5-Aminoimidazole-4-Carboxamide Ribonucleoside. A Specific Method for Activating AMP-Activated Protein Kinase in Intact Cells?

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