Renal Dopamine Receptor Signaling Mechanisms in Spontaneously Hypertensive and Fischer 344 Old Rats
Author:
Publisher
Informa UK Limited
Subject
General Medicine,Physiology,Internal Medicine
Link
http://www.tandfonline.com/doi/pdf/10.3109/10641969909068646
Reference51 articles.
1. Defective dopamine-1 receptor adenylate cyclase coupling in the proximal convoluted tubule from the spontaneously hypertensive rat.
2. Role of the D1A dopamine receptor in the pathogenesis of genetic hypertension.
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2. Instrumental conditioning for food reinforcement in the spontaneously hypertensive rat model of attention deficit hyperactivity disorder;BMC Research Notes;2017-10-30
3. Loss of NHERF-1 expression prevents dopamine-mediated Na-K-ATPase regulation in renal proximal tubule cells from rat models of hypertension: aged F344 rats and spontaneously hypertensive rats;American Journal of Physiology-Cell Physiology;2017-08-01
4. Downregulation of Renal G Protein–Coupled Receptor Kinase Type 4 Expression via Ultrasound‐Targeted Microbubble Destruction Lowers Blood Pressure in Spontaneously Hypertensive Rats;Journal of the American Heart Association;2016-10-03
5. Appetitive and consummative responding for liquid sucrose in the spontaneously hypertensive rat model of attention deficit hyperactivity disorder;Behavioural Brain Research;2013-02
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