Is there a role for JAK inhibitors inBCR-ABL1-negative myeloproliferative neoplasms other than myelofibrosis?
Author:
Publisher
Informa UK Limited
Subject
Cancer Research,Oncology,Hematology
Link
http://www.tandfonline.com/doi/pdf/10.3109/10428194.2014.985159
Reference62 articles.
1. Integrated genomic analysis illustrates the central role of JAK-STAT pathway activation in myeloproliferative neoplasm pathogenesis
2. A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera
3. MPLW515L Is a Novel Somatic Activating Mutation in Myelofibrosis with Myeloid Metaplasia
4. MPL515 mutations in myeloproliferative and other myeloid disorders: a study of 1182 patients
5. JAK2Exon 12 Mutations in Polycythemia Vera and Idiopathic Erythrocytosis
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1. Molecular Pathogenesis of BCR-ABL-Negative Atypical Chronic Myeloid Leukemia;Frontiers in Oncology;2021-11-11
2. Targeting few to help hundreds: JAK, MAPK and ROCK pathways as druggable targets in atypical chronic myeloid leukemia;Molecular Cancer;2018-02-19
3. Latent Myeloproliferative Neoplasm as a Cause of Idiopathic Splanchnic Vein Thrombosis: The Role of JAK2V617F Mutation and Bone Marrow Biopsy Evaluation;Hematology & Transfusion International Journal;2015-11-18
4. Triggering of Suicidal Erythrocyte Death by Ruxolitinib;Cellular Physiology and Biochemistry;2015
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