ConcomitantFIP1L1–PDGFRAfusion gene and T-cell clonality in a case of chronic eosinophilic leukemia with clonal evolution and an incomplete response to imatinib-Reference-Cited by-同舟云学术

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ConcomitantFIP1L1–PDGFRAfusion gene and T-cell clonality in a case of chronic eosinophilic leukemia with clonal evolution and an incomplete response to imatinib

Published:2011-02 Issue:2 Volume:52 Page:335-338
ISSN:1042-8194
Container-title:Leukemia & Lymphoma
language:en
Short-container-title:Leukemia & Lymphoma

Author:

Burbury Kate,Chew Lee Ping,Westerman David,Catalano Alberto,Seymour John F.

Publisher

Informa UK Limited

Subject

Cancer Research,Oncology,Hematology

Link

http://www.tandfonline.com/doi/pdf/10.3109/10428194.2010.534210

Reference15 articles.

1. A Tyrosine Kinase Created by Fusion of thePDGFRAandFIP1L1Genes as a Therapeutic Target of Imatinib in Idiopathic Hypereosinophilic Syndrome

2. Molecular profiling of CD3−CD4+ T cells from patients with the lymphocytic variant of hypereosinophilic syndrome reveals targeting of growth control pathways

3. T-cell abnormalities are present at high frequencies in patients with hypereosinophilic syndrome

Cited by 4 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Case of polycythemia vera concurrent with FIP1L1-PDGFRA–positive myeloproliferative neoplasm with eosinophilia;Cancer Genetics;2012-10

2. Identification of JAK2 as a Mediator of FIP1L1-PDGFRA-Induced Eosinophil Growth and Function in CEL;PLoS ONE;2012-04-16

3. Therapeutic Approaches to Patients With Hypereosinophilic Syndromes;Seminars in Hematology;2012-04

4. Eosinophilic Myeloproliferative Disorders;Hematology;2011-12-10

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