Novel therapies for myelofibrosis
Author:
Publisher
Informa UK Limited
Subject
Cancer Research,Oncology,Hematology
Link
http://www.tandfonline.com/doi/pdf/10.3109/10428194.2015.1037762
Reference107 articles.
1. Comparison of placebo and best available therapy for the treatment of myelofibrosis in the phase 3 COMFORT studies
2. A unique clonal JAK2 mutation leading to constitutive signalling causes polycythaemia vera
3. A Gain-of-Function Mutation ofJAK2in Myeloproliferative Disorders
4. Activating mutation in the tyrosine kinase JAK2 in polycythemia vera, essential thrombocythemia, and myeloid metaplasia with myelofibrosis
Cited by 6 articles. 订阅此论文施引文献 订阅此论文施引文献,注册后可以免费订阅5篇论文的施引文献,订阅后可以查看论文全部施引文献
1. HDAC11 deficiency disrupts oncogene-induced hematopoiesis in myeloproliferative neoplasms;Blood;2020-01-16
2. Janus kinase-2 inhibitor fedratinib in patients with myelofibrosis previously treated with ruxolitinib (JAKARTA-2): a single-arm, open-label, non-randomised, phase 2, multicentre study;The Lancet Haematology;2017-07
3. Pacritinib versus best available therapy for the treatment of myelofibrosis irrespective of baseline cytopenias (PERSIST-1): an international, randomised, phase 3 trial;The Lancet Haematology;2017-05
4. The role of the extracellular matrix in primary myelofibrosis;Blood Cancer Journal;2017-02
5. Primary analysis of a phase II open-label trial of INCB039110, a selective JAK1 inhibitor, in patients with myelofibrosis;Haematologica;2016-10-27
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