tp53-dependent G2 arrest mediator candidate gene,Reprimo, is down-regulated by promoter hypermethylation in pediatric acute myeloid leukemia
Author:
Publisher
Informa UK Limited
Subject
Cancer Research,Oncology,Hematology
Link
http://www.tandfonline.com/doi/pdf/10.3109/10428194.2015.1011157
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1. Two regression models and a scoring system for predicting survival and planning treatment in myelodysplastic syndromes: a multivariate analysis of prognostic factors in 370 patients
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1. Reprimo (RPRM) mediates neuronal ferroptosis via CREB-Nrf2/SCD1 pathways in radiation-induced brain injury;Free Radical Biology and Medicine;2024-03
2. RPRM deletion preserves hematopoietic regeneration by promoting EGFR‐dependent DNA repair and hematopoietic stem cell proliferation post ionizing radiation;Cell Biology International;2022-08-30
3. DNA Methylation Clusters and Their Relation to Cytogenetic Features in Pediatric AML;Cancers;2020-10-17
4. The Reprimo gene family member, reprimo-like (rprml), is required for blood development in embryonic zebrafish;Scientific Reports;2019-05-09
5. Molecular mechanism of G1 arrest and cellular senescence induced by LEE011, a novel CDK4/CDK6 inhibitor, in leukemia cells;Cancer Cell International;2017-03-06
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