Bepridil Exacerbates Glutamate-Induced Deterioration of Calcium Homeostasis and Cultured Nerve Cell Injury-Reference-Cited by-同舟云学术

Bepridil Exacerbates Glutamate-Induced Deterioration of Calcium Homeostasis and Cultured Nerve Cell Injury

Published:1996-01 Issue:3-4 Volume:88 Page:199-214
ISSN:0020-7454
Container-title:International Journal of Neuroscience
language:en
Short-container-title:International Journal of Neuroscience

Author:

Storozhevykh Tatyana P.,Sorokina Elena G.,Vinskaya Natalya P.,Pinelis Vsevolod G.,Vergun Olga V.,Fayuk Dmitriy A.,Sobolevskiy Alexander I.,Khodorov Boris I.

Publisher

Informa UK Limited

Subject

General Medicine,General Neuroscience

Link

http://www.tandfonline.com/doi/pdf/10.3109/00207459609000615

Reference24 articles.

1. Inhibition of Na+/Ca2+ exchange enhances delayed neuronal death elicited by glutamate in cerebellar granule cell cultures

2. Electrophysiology of glutamate neurotoxicity in vitro: induction of a calcium-dependent extended neuronal depolarization

3. Intracellular calcium concentrations during "chemical hypoxia" and excitotoxic neuronal injury

4. Inhibition of sodium-calcium exchange in cardiac sarcolemmal membrane vesicles. 2. Mechanism of inhibition by bepridil

5. A new generation of Ca2+ indicators with greatly improved fluorescence properties.

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1. Na+/Ca2+ exchange and regulation of cytoplasmic concentration of calcium in rat cerebellar neurons treated with glutamate;Biochemistry (Moscow);2007-07

2. Neuronal preconditioning with the antianginal drug, bepridil;Journal of Neurochemistry;2007-02-26

3. The influence of sodium–calcium exchange inhibitors on rabbit lens ion balance and transparency;Experimental Eye Research;2006-11

4. Angiotensin type 2 receptor neuroprotection against chemical hypoxia is dependent on the delayed rectifier K+ channel, Na+/Ca2+ exchanger and Na+/K+ ATPase in primary cortical cultures;Neuroscience Research;2004-11

5. Calcium influx constitutes the ionic basis for the maintenance of glutamate-induced extended neuronal depolarization associated with hippocampal neuronal death;Cell Calcium;2003-02