Fibroblast growth factor-2-induced cardioprotection against myocardial infarction occurs via the interplay between nitric oxide, protein kinase signaling, and ATP-sensitive potassium channels-Reference-Cited by-同舟云学术

Fibroblast growth factor-2-induced cardioprotection against myocardial infarction occurs via the interplay between nitric oxide, protein kinase signaling, and ATP-sensitive potassium channels

Published:2012-02-06 Issue:2 Volume:30 Page:124-139
ISSN:0897-7194
Container-title:Growth Factors
language:en
Short-container-title:Growth Factors

Author:

Manning Janet R.,Carpenter Gregory,Porter Darius R.,House Stacey L.,Pietras Daniel A.,Doetschman Thomas,Schultz Jo El J.

Publisher

Informa UK Limited

Subject

Cell Biology,Clinical Biochemistry,Endocrinology

Link

http://www.tandfonline.com/doi/pdf/10.3109/08977194.2012.656759

Reference96 articles.

1. Neuronal Nitric-oxide Synthase Mutant (Ser-1412 → Asp) Demonstrates Surprising Connections between Heme Reduction, NO Complex Formation, and Catalysis

2. Nitric oxide synthase isoform inhibition before whole body ischemia reperfusion in pigs: Vital or protective?

3. Cilostazol protects the heart against ischaemia reperfusion injury in a rabbit model of myocardial infarction: Focus on adenosine, nitric oxide and mitochondrial ATP-sensitive potassium channels

4. Protein Kinase Cε Interacts With and Inhibits the Permeability Transition Pore in Cardiac Mitochondria

5. Inhibition of the Cardiac p38-MAPK Pathway by SB203580 Delays Ischemic Cell Death

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