Heterozygous ITGA2B Phe1024 Deletion Associated with Abnormal αIIbβ3 Function in a Patient with Congenital Thrombocytopenia
Author:
Affiliation:
1. Department of Laboratory Medicine, Tongji Hospital, Tongji Medical College, Huazhong University of Science and Technology, Wuhan, China
Publisher
Georg Thieme Verlag KG
Link
http://www.thieme-connect.de/products/ejournals/pdf/10.1055/s-0044-1785655.pdf
Reference16 articles.
1. αIIbβ3: structure and function;B S Coller;J Thromb Haemost,2015
2. Platelet integrin αIIbβ3: signal transduction, regulation, and its therapeutic targeting;J Huang;J Hematol Oncol,2019
3. Glanzmann thrombasthenia: state of the art and future directions;A T Nurden;Semin Thromb Hemost,2013
4. Glanzmann thrombasthenia: a review of ITGA2B and ITGB3 defects with emphasis on variants, phenotypic variability, and mouse models;A T Nurden;Blood,2011
5. Congenital macrothrombocytopenias;S Kunishima;Blood Rev,2006
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