Fibroblast Growth Factor 23, Klotho, and Disordered Mineral Metabolism in Chronic Kidney Disease: Unraveling the Intricate Tapestry of Events and Implications for Therapy
Author:
Publisher
Elsevier BV
Subject
Nephrology,Nutrition and Dietetics,Medicine (miscellaneous)
Reference36 articles.
1. Skeletal secretion of FGF-23 regulates phosphate and vitamin D metabolism;Quarles;Nat Rev Endocrinol,2012
2. Human fibroblast growth factor-23 mutants suppress Na+-dependent phosphate co-transport activity and 1alpha,25-dihydroxyvitamin D3 production;Saito;J Biol Chem,2003
3. FGF-23 inhibits renal tubular phosphate transport and is a PHEX substrate;Bowe;Biochem Biophys Res Commun,2001
4. Fibroblast growth factor 23 is a counter-regulatory phosphaturic hormone for vitamin D;Liu;J Am Soc Nephrol,2006
5. The parathyroid is a target organ for FGF23 in rats;Ben-Dov;J Clin Invest,2007
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1. Analysis of the association between serum antiaging humoral factor klotho and cardiovascular disease potential risk factor apolipoprotein B in general population;Medicine;2023-06-23
2. 1,25-dihydroxyvitamin D as Predictor of Renal Worsening Function in Chronic Kidney Disease. Results From the PASCaL-1,25D Study;Frontiers in Medicine;2022-03-02
3. Toxines urémiques de moyen poids moléculaire : un véritable regain d’intérêt;Néphrologie & Thérapeutique;2019-04
4. X-Linked Hypophosphatemia and FGF23-Related Hypophosphatemic Diseases: Prospect for New Treatment;Endocrine Reviews;2018-01-26
5. Recombinant α-Klotho may be prophylactic and therapeutic for acute to chronic kidney disease progression and uremic cardiomyopathy;Kidney International;2017-05
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