Intestinal Hyperuricemia as a Driving Mechanism for CKD
Author:
Funder
Horizon Therapeutics
National Institutes of Health
Publisher
Elsevier BV
Subject
Nephrology
Reference28 articles.
1. Effects of allopurinol on the progression of chronic kidney disease;Badve;N Engl J Med,2020
2. Urate transporter ABCG2 function and asymptomatic hyperuricemia: a retrospective cohort study of CKD progression;Ohashi;Am J Kidney Dis,2023
3. Decreased extra-renal urate excretion is a common cause of hyperuricemia;Ichida;Nat Commun,2012
4. Early-onset metabolic syndrome in mice lacking the intestinal uric acid transporter SLC2A9;DeBosch;Nat Commun,2014
5. ABCG2 dysfunction increases serum uric acid by decreased intestinal urate excretion;Takada;Nucleosides Nucleotides Nucleic Acids,2014
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1. 9-Hydroxy-8-oxypalmatine, a novel liver-mediated oxymetabolite of palmatine, alleviates hyperuricemia and kidney inflammation in hyperuricemic mice;Journal of Ethnopharmacology;2024-12
2. Imperata cylindrica polysaccharide ameliorates intestinal dysbiosis and damage in hyperuricemic nephropathy;International Journal of Biological Macromolecules;2024-10
3. Hyperuricemia and its related diseases: mechanisms and advances in therapy;Signal Transduction and Targeted Therapy;2024-08-28
4. Allopurinol, Febuxostat, and Nonuse of Xanthine Oxidoreductase Inhibitor Treatment in Patients Receiving Hemodialysis: A Longitudinal Analysis;Kidney Medicine;2024-08
5. Association between urinary uric acid excretion and kidney outcome in patients with CKD;Scientific Reports;2024-03-01
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