A “Two-in-One Hit” Model of Shortcut Carcinogenesis in MLH1 Lynch Syndrome Carriers
Author:
Publisher
Elsevier BV
Subject
Gastroenterology,Hepatology
Reference13 articles.
1. Associations of Pathogenic Variants in MLH1, MSH2, and MSH6 With Risk of Colorectal Adenomas and Tumors and With Somatic Mutations in Patients With Lynch Syndrome
2. Three molecular pathways model colorectal carcinogenesis in Lynch syndrome
3. WNT signalling pathways as therapeutic targets in cancer
4. Correction to: The majority of β-catenin mutations in colorectal cancer is homozygous
5. Assessment of Tumor Sequencing as a Replacement for Lynch Syndrome Screening and Current Molecular Tests for Patients With Colorectal Cancer
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1. Lower degree of microsatellite instability in colorectal carcinomas fromMSH6-associated Lynch syndrome patients;2024-08-12
2. Gastrointestinal Cancer Precursor Conditions and Their Detection;Hematology/Oncology Clinics of North America;2024-08
3. Incidences of colorectal adenomas and cancers under colonoscopy surveillance suggest an accelerated “Big Bang” pathway to CRC in three of the four Lynch syndromes;Hereditary Cancer in Clinical Practice;2024-05-13
4. Genomic Landscape of Lynch Syndrome Colorectal Neoplasia Identifies Shared Mutated Neoantigens for Immunoprevention;Gastroenterology;2024-05
5. Mitotic abnormalities precede microsatellite instability in lynch syndrome-associated colorectal tumourigenesis;eBioMedicine;2024-05
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