Fatty Acid Ethyl Esters Cause Pancreatic Calcium Toxicity via Inositol Trisphosphate Receptors and Loss of ATP Synthesis-Reference-Cited by-同舟云学术

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Fatty Acid Ethyl Esters Cause Pancreatic Calcium Toxicity via Inositol Trisphosphate Receptors and Loss of ATP Synthesis

Published:2006-03 Issue:3 Volume:130 Page:781-793
ISSN:0016-5085
Container-title:Gastroenterology
language:en
Short-container-title:Gastroenterology

Author:

Criddle David N.,Murphy John,Fistetto Gregorio,Barrow Stephanie,Tepikin Alexei V.,Neoptolemos John P.,Sutton Robert,Petersen Ole H.

Publisher

Elsevier BV

Subject

Gastroenterology,Hepatology

Reference51 articles.

1. Course of alcoholic chronic pancreatitis: a prospective clinicomorphological long-term study;Ammann;Gastroenterology,1996

2. Presence of nonoxidative ethanol metabolism in human organs commonly damaged by ethanol abuse;Laposata;Science,1986

3. Ethanol metabolism and transcription factor activation in pancreatic acinar cells in rats;Gukovskaya;Gastroenterology,2002

4. Characterization of enzymes involved in formation of ethyl esters of long-chain fatty acids in humans;Diczfalusy;J Lipid Res,2001

5. Pancreatic injury in rats induced by fatty acid ethyl ester, a nonoxidative metabolite of alcohol;Werner;Gastroenterology,1997

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