Evolution of a chemosensitive core-binding factor AML into an aggressive leukemia with eosinophilic differentiation-Reference-Cited by-同舟云学术

Evolution of a chemosensitive core-binding factor AML into an aggressive leukemia with eosinophilic differentiation

Published:2018-06-27 Issue:13 Volume:2 Page:1517-1521
ISSN:2473-9529
Container-title:Blood Advances
language:en
Short-container-title:

Author:

Xiao Wenbin¹^ORCID,Yabe Mariko¹,Offin Michael²,Khattar Pallavi¹,Baik Jeeyeon¹,Daley Ryan J.²,Pappacena Jeremy J.²,Roshal Mikhail¹,Zhang Yanming¹,Tallman Martin S.²,Cai Sheng F.²^ORCID

Affiliation:

1. Hematopathology Diagnostic Service, Department of Pathology, and

2. Leukemia Service, Department of Medicine, Memorial Sloan Kettering Cancer Center, New York, NY

Abstract

Key Points Core-binding factor AML can evolve from good-risk disease into aggressive disease through the gain of additional genomic aberrations. In this unique case, an AML patient died of hypereosinophilic syndrome with solid organ infiltration of differentiated eosinophils.

Publisher

American Society of Hematology

Subject

Hematology

Link

http://ashpublications.org/bloodadvances/article-pdf/2/13/1517/881252/advances016675.pdf

Reference10 articles.

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3. The abnormal eosinophils are part of the leukemic cell population in acute myelomonocytic leukemia with abnormal eosinophils (AML M4Eo) and carry the pericentric inversion 16: a combination of May-Grünwald-Giemsa staining and fluorescence in situ hybridization;Haferlach;Blood,1996

4. Abnormal chromosome 16 in clonogenic eosinophils from a case of acute myeloid leukemia (FAB,M2);Hollings;Cancer Genet Cytogenet,1988

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