Platelets mediate increased endothelium permeability in dengue through NLRP3-inflammasome activation-Reference-Cited by-同舟云学术

Platelets mediate increased endothelium permeability in dengue through NLRP3-inflammasome activation

Published:2013-11-14 Issue:20 Volume:122 Page:3405-3414
ISSN:0006-4971
Container-title:Blood
language:en
Short-container-title:

Author:

Hottz Eugenio D.¹²,Lopes Juliana F.¹,Freitas Carla¹,Valls-de-Souza Rogério²,Oliveira Marcus F.³,Bozza Marcelo T.⁴,Da Poian Andrea T.³,Weyrich Andrew S.⁵⁶,Zimmerman Guy A.¹⁵,Bozza Fernando A.²,Bozza Patricia T.¹

Affiliation:

1. Laboratório de Immunofarmacologia, Instituto Oswaldo Cruz, and

2. Instituto de Pesquisa Clinica Evandro Chagas, Fundação Oswaldo Cruz, Rio de Janeiro, Brazil;

3. Instituto de Bioquímica Médica and

4. Instituto de Microbiologia, Universidade Federal do Rio de Janeiro, Rio de Janeiro, Brazil; and

5. Department of Medicine and

6. Molecular Medicine Program, University of Utah, Salt Lake City, UT

Abstract

Key PointsDengue infection triggers functional inflammasome assembly in platelets. Platelets may contribute to increased vascular permeability in dengue virus infection by synthesis and release of IL-1β.

Publisher

American Society of Hematology

Subject

Cell Biology,Hematology,Immunology,Biochemistry

Link

http://ashpublications.org/blood/article-pdf/122/20/3405/1371699/3405.pdf

Reference50 articles.

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4. Multiplex cytokine profile from dengue patients: MIP-1beta and IFN-gamma as predictive factors for severity.;Bozza;BMC Infect Dis,2008

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