LNK mutations in familial myeloproliferative neoplasms-Reference-Cited by-同舟云学术

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LNK mutations in familial myeloproliferative neoplasms

Published:2016-07-07 Issue:1 Volume:128 Page:144-145
ISSN:0006-4971
Container-title:Blood
language:en
Short-container-title:

Author:

Rumi Elisa¹²^ORCID,Harutyunyan Ashot S.³,Pietra Daniela¹,Feenstra Jelena D. Milosevic³,Cavalloni Chiara²,Roncoroni Elisa¹,Casetti Ilaria²,Bellini Marta²,Milanesi Chiara¹,Renna Maria C.¹,Gotti Manuel¹,Astori Cesare¹,Kralovics Robert³,Cazzola Mario¹²

Affiliation:

1. Department of Hematology Oncology, Fondazione Istituto di Ricovero e Cura a Carattere Scientifico Policlinico San Matteo, Pavia, Italy;

2. Department of Molecular Medicine, University of Pavia, Pavia, Italy; and

3. CeMM Research Center for Molecular Medicine of the Austrian Academy of Sciences, Vienna, Austria

Publisher

American Society of Hematology

Subject

Cell Biology,Hematology,Immunology,Biochemistry

Link

http://ashpublications.org/blood/article-pdf/128/1/144/1394977/144.pdf

Reference14 articles.

1. Genetic and clinical implications of the Val617Phe JAK2 mutation in 72 families with myeloproliferative disorders.;Bellanné-Chantelot;Blood,2006

2. Familial chronic myeloproliferative disorders: clinical phenotype and evidence of disease anticipation.;Rumi;J Clin Oncol,2007

3. CALR exon 9 mutations are somatically acquired events in familial cases of essential thrombocythemia or primary myelofibrosis.;Rumi;Blood,2014

4. JAK2 (V617F) as an acquired somatic mutation and a secondary genetic event associated with disease progression in familial myeloproliferative disorders.;Rumi;Cancer,2006

5. A common JAK2 haplotype confers susceptibility to myeloproliferative neoplasms.;Olcaydu;Nat Genet,2009

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