The orphan nuclear receptor TR4 regulates erythroid cell proliferation and maturation-Reference-Cited by-同舟云学术

The orphan nuclear receptor TR4 regulates erythroid cell proliferation and maturation

Published:2017-12-07 Issue:23 Volume:130 Page:2537-2547
ISSN:0006-4971
Container-title:Blood
language:en
Short-container-title:

Author:

Lee Mary P.¹,Tanabe Osamu¹²,Shi Lihong¹,Jearawiriyapaisarn Natee¹³^ORCID,Lucas Daniel¹,Engel James Douglas¹

Affiliation:

1. Department of Cell and Developmental Biology, University of Michigan Medical School, Ann Arbor, MI;

2. Department of Integrative Genomics, Tohoku Medical Megabank, Tohoku University, Sedai, Japan; and

3. Thalassemia Research Center, Institute of Molecular Biosciences, Mahidol University, Salaya, Nakhon Pathom, Thailand

Abstract

Key Points TR4 and TR2 execute distinct functions during embryogenesis and erythroid differentiation. TR4 promotes erythroid proliferation and maturation.

Publisher

American Society of Hematology

Subject

Cell Biology,Hematology,Immunology,Biochemistry

Link

http://ashpublications.org/blood/article-pdf/130/23/2537/1404068/blood783159.pdf

Reference58 articles.

1. Nuclear receptors TR2 and TR4 recruit multiple epigenetic transcriptional corepressors that associate specifically with the embryonic β-type globin promoters in differentiated adult erythroid cells;Cui;Mol Cell Biol,2011

2. The TR2 and TR4 orphan nuclear receptors repress Gata1 transcription;Tanabe;Genes Dev,2007

3. Embryonic and fetal beta-globin gene repression by the orphan nuclear receptors, TR2 and TR4;Tanabe;EMBO J,2007

4. An embryonic/fetal beta-type globin gene repressor contains a nuclear receptor TR2/TR4 heterodimer;Tanabe;EMBO J,2002

5. Compound loss of function of nuclear receptors Tr2 and Tr4 leads to induction of murine embryonic β-type globin genes;Cui;Blood,2015

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