Inflammation is key to hemophilic arthropathy
Author:
Affiliation:
1. CHRISTIAN MEDICAL COLLEGE, VELLORE
Publisher
American Society of Hematology
Subject
Cell Biology,Hematology,Immunology,Biochemistry
Link
http://ashpublications.org/blood/article-pdf/126/19/2175/1345123/2175.pdf
Reference11 articles.
1. IL-1β, in contrast to TNFα, is pivotal in blood-induced cartilage damage and is a potential target for therapy.;van Vulpen,2015
2. Guidelines for the management of hemophilia.;Srivastava;Haemophilia,2013
3. Prophylaxis versus episodic treatment to prevent joint disease in boys with severe hemophilia.;Manco-Johnson;N Engl J Med,2007
4. Haemoglobin-derived iron-dependent hydroxyl radical formation in blood-induced joint damage: an in vitro study.;Hooiveld;Rheumatology (Oxford),2003
5. IL-4 alone and in combination with IL-10 protects against blood-induced cartilage damage.;van Meegeren;Osteoarthritis Cartilage,2012
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1. Aberrant methylation and expression of TNXB promotes chondrocyte apoptosis and extracullar matrix degradation in hemophilic arthropathy via AKT signaling;2023-12-11
2. IL-6, IL-1β and TNF-α regulation of the chondrocyte phenotype: a possible mechanism of haemophilic cartilage destruction;Hematology;2023-02-17
3. Cinnamic aldehyde, an anti-inflammatory component in Du-Huo-Ji-Sheng-Tang, ameliorates arthritis in II collagenase and monosodium iodoacetate induced osteoarthritis rat models;Journal of Traditional and Complementary Medicine;2023-01
4. FGF23 and SOX9 expression in haemophilic cartilage: In vitro studies of the effects of iron;Haemophilia;2022-07-08
5. The Application of Focused Medium-Energy Extracorporeal Shockwave Therapy in Hemophilic A Arthropathy;Healthcare;2022-02-11
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