Concomitant occurrence of BCR-ABL and JAK2V617F mutation-Reference-Cited by-同舟云学术

Concomitant occurrence of BCR-ABL and JAK2V617F mutation

Published:2011-09-22 Issue:12 Volume:118 Page:3445-3446
ISSN:0006-4971
Container-title:Blood
language:en
Short-container-title:

Author:

Pieri Lisa¹,Spolverini Ambra¹,Scappini Barbara¹,Occhini Umberto²,Birtolo Silvia³,Bosi Alberto¹,Albano Francesco⁴,Fava Carmen⁵,Vannucchi Alessandro M.¹

Affiliation:

1. Section of Hematology, Department of Critical Care, University of Florence, Florence, Italy

2. Section of Hematology, S. Donato Hospital, Arezzo, Italy

3. Medical Oncology ASL 3, Pistoia, Italy

4. Hematology Institute, University of Bari, Bari, Italy

5. Department of Clinical and Biological Sciences, University of Turin, Orbassano, Italy

Publisher

American Society of Hematology

Subject

Cell Biology,Hematology,Immunology,Biochemistry

Link

http://ashpublications.org/blood/article-pdf/118/12/3445/1340381/zh803811003445.pdf

Reference5 articles.

1. CBL, CBLB, TET2, ASXL1, and IDH1/2 mutations and additional chromosomal aberrations constitute molecular events in chronic myelogenous leukemia.;Makishima;Blood,2011

2. Chronic myeloproliferative diseases with concurrent BCR-ABL junction and JAK2V617F mutation.;Hussein;Leukemia,2008

3. Establishment of the first World Health Organization International Genetic Reference Panel for quantitation of BCR-ABL mRNA.;White;Blood,2010

4. Identification of patients with poorer survival in primary myelofibrosis based on the burden of JAK2V617F mutated allele.;Guglielmelli;Blood,2009

5. Insights into JAK2-V617F mutation in CML.;Bocchia;Lancet Oncol,2007

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2. Co-occurrence of JAK2-V617 F mutation and BCR::ABL1 translocation in chronic myeloproliferative neoplasms: a potentially confounding genetic combination;Frontiers in Oncology;2024-01-12

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4. Chronic myeloid leukemia (CML) evolves from Philadelphia chromosome-negative myeloproliferative neoplasms (MPNs) with unexpected frequency;International Journal of Hematology;2022-10-01

5. JAK2 V617f in chronic myeloid leukemia: driving force or passive bystander?;Hematology;2022-08-08