p14ARF,p15INK4b, and p16INK4amethylation status in chronic myelogenous leukemia

Author:

Kusy Sophie1,Cividin Marie1,Sorel Nathalie1,Brizard Françoise1,Guilhot François1,Brizard André1,Larsen Christian1,Roche Joëlle1

Affiliation:

1. Correspondence: Joëlle Roche, EA 2224, IBMIG, Université de Poitiers, 40 avenue du Recteur Pineau, 86022 Poitiers Cédex, France; e-mail:

Publisher

American Society of Hematology

Subject

Cell Biology,Hematology,Immunology,Biochemistry

Reference9 articles.

1. DNA methylation patterns in hereditary human cancers mimic sporadic tumorigenesis.;Esteller;Hum Mol Genet.,2001

2. Distinct patterns of inactivation of p15INK4B and p16INK4A characterize the major types of hematological malignancies.;Herman;Cancer Res.,1997

3. Quantitative measure of c-abl and p15 methylation in chronic myelogenous leukemia: biological implications.;Nguyen;Blood.,2000

4. Methylation-specific PCR: a novel PCR assay for methylation status of CpG islands.;Herman;Proc Natl Acad Sci U S A.,1996

5. Interferon alfa-2b combined with cytarabine versus interferon alone in chronic myelogenous leukemia.;Guilhot;N Engl J Medicine.,1997

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