SRF is required for neutrophil migration in response to inflammation-Reference-Cited by-同舟云学术

SRF is required for neutrophil migration in response to inflammation

Published:2014-05-08 Issue:19 Volume:123 Page:3027-3036
ISSN:0006-4971
Container-title:Blood
language:en
Short-container-title:

Author:

Taylor Ashley¹,Tang Wenwen²,Bruscia Emanuela M.³⁴,Zhang Ping-Xia³,Lin Aiping³,Gaines Peter⁵,Wu Dianqing²,Halene Stephanie¹

Affiliation:

1. Section of Hematology, Department of Internal Medicine and Yale Comprehensive Cancer Center,

2. Department of Pharmacology, Vascular Biology and Therapeutics Program, and

3. Department of Pediatrics, Yale University School of Medicine, New Haven, CT;

4. European Institute for Research in Cystic Fibrosis, San Raffaele Scientific Institute, Milan, Italy; and

5. Department of Biological Science, Biomedical Engineering and Biotechnology, University of Massachusetts, Lowell, MA

Abstract

Key Points SRF is essential for neutrophil migration in part by regulation of integrin homeostasis. Several genes located on chromosome 5q are part of the SRF signaling pathway implicating dysfunction of SRF in myelodysplasia.

Publisher

American Society of Hematology

Subject

Cell Biology,Hematology,Immunology,Biochemistry

Link

http://ashpublications.org/blood/article-pdf/123/19/3027/1376558/3027.pdf

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4. Differential usage of signal transduction pathways defines two types of serum response factor target gene.;Gineitis;J Biol Chem,2001

5. Identification of miR-145 and miR-146a as mediators of the 5q- syndrome phenotype.;Starczynowski;Nat Med,2010

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