HIFs: a-cute answer to inflammation?
Author:
Affiliation:
1. THE OHIO STATE UNIVERSITY
Publisher
American Society of Hematology
Subject
Cell Biology,Hematology,Immunology,Biochemistry
Link
http://ashpublications.org/blood/article-pdf/118/3/485/1346548/zh802911000485.pdf
Reference8 articles.
1. Activation of Hif-1alpha delays inflammation resolution by reducing neutrophil apoptosis and reverse migration in a zebrafish inflammation model.;Elks;Blood,2011
2. Interleukin-1beta and tumor necrosis factor-alpha stimulate DNA binding of hypoxia-inducible factor-1.;Hellwig-Bürgel;Blood,1999
3. Vascular endothelial growth factor (VEGF) induces remodeling and enhances TH2-mediated sensitization and inflammation in the lung.;Lee;Nat Med,2004
4. Resolution of inflammation: the beginning programs the end.;Serhan;Nat Immunol,2005
5. Oxygen-independent stabilization of hypoxia inducible factor (HIF)-1 during RSV infection.;Haeberle;PLoS ONE,2008
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3. Expression of hypoxia-inducible factor-1α (HIF-1α) in infiltrating inflammatory cells is associated with chronic allograft dysfunction and predicts long-term graft survival;Nephrology Dialysis Transplantation;2012-09-30
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