The JAK-STAT signaling pathway is differentially activated in CALR-positive compared with JAK2V617F-positive ET patients-Reference-Cited by-同舟云学术

The JAK-STAT signaling pathway is differentially activated in CALR-positive compared with JAK2V617F-positive ET patients

Published:2015-03-05 Issue:10 Volume:125 Page:1679-1681
ISSN:0006-4971
Container-title:Blood
language:en
Short-container-title:

Author:

Lau Winnie W. Y.¹^ORCID,Hannah Rebecca²,Green Anthony R.³,Göttgens Bertie¹^ORCID

Affiliation:

1. Cambridge Institute for Medical Research and Wellcome Trust/Medical Research Council, Stem Cell Institute and Department of Haematology, University of Cambridge, Cambridge, United Kingdom

2. Cambridge Institute for Medical Research and Wellcome Trust/Medical Research Council, Stem Cell Institute and Department of Haematology, University of Cambridge, Cambridge United Kingdom

3. Cambridge Institute for Medical Research and Wellcome Trust/Medical Research Council, Stem Cell Institute and Department of Haematology, University of Cambridge, Cambridge, United Kingdom Department of Haematology, Addenbrooke’s Hospital, Cambridge, United Kingdom

Publisher

American Society of Hematology

Subject

Cell Biology,Hematology,Immunology,Biochemistry

Link

http://ashpublications.org/blood/article-pdf/125/10/1679/1382604/1679.pdf

Reference10 articles.

1. Myeloproliferative neoplasms: JAK2 signaling pathway as a central target for therapy.;Pasquier;Clin Lymphoma Myeloma Leuk,2014

2. Janus kinase deregulation in leukemia and lymphoma.;Chen;Immunity,2012

3. Somatic mutations of calreticulin in myeloproliferative neoplasms.;Klampfl;N Engl J Med,2013

4. Somatic CALR mutations in myeloproliferative neoplasms with nonmutated JAK2.;Nangalia;N Engl J Med,2013

5. MARIMO cells harbor a CALR mutation but are not dependent on JAK2/STAT5 signaling [letter].;Kollmann;Leukemia,2015

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