Epigenetic silencing of miR-125b is required for normal B-cell development-Reference-Cited by-同舟云学术

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Epigenetic silencing of miR-125b is required for normal B-cell development

Published:2018-04-26 Issue:17 Volume:131 Page:1920-1930
ISSN:0006-4971
Container-title:Blood
language:en
Short-container-title:

Author:

Li Guideng¹,So Alex Yick-Lun¹,Sookram Reeshelle¹,Wong Stephanie¹,Wang Jessica K.¹,Ouyang Yong¹,He Peng¹,Su Yapeng²,Casellas Rafael³⁴,Baltimore David¹

Affiliation:

1. Division of Biology and Biological Engineering and

2. Division of Chemistry and Chemical Engineering, California Institute of Technology, Pasadena, CA; and

3. Lymphocyte Nuclear Biology, National Institute of Arthritis and Musculoskeletal and Skin Diseases, and

4. Center of Cancer Research, National Cancer Institute, National Institutes of Health, Bethesda, MD

Abstract

Key Points miR-125b is epigenetically silenced in B cells. Physiological silencing of miR-125b is required for normal B-cell development.

Publisher

American Society of Hematology

Subject

Cell Biology,Hematology,Immunology,Biochemistry

Link

http://ashpublications.org/blood/article-pdf/131/17/1920/1406310/blood824540.pdf

Reference26 articles.

1. Transcription control of early B cell differentiation;Mandel;Curr Opin Immunol,2010

2. S1P1 receptor directs the release of immature B cells from bone marrow into blood;Allende;J Exp Med,2010

3. CXCR4 and a cell-extrinsic mechanism control immature B lymphocyte egress from bone marrow;Beck;J Exp Med,2014

4. Cannabinoid receptor 2 mediates the retention of immature B cells in bone marrow sinusoids;Pereira;Nat Immunol,2009

5. A role for S1P and S1P1 in immature-B cell egress from mouse bone marrow;Pereira;PLoS One,2010

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4. Assessment of mechanism of Ganoderma lucidum neutral triterpenes mediating bone marrow mesenchymal stem cells with high expression of miR-125b targeting Bak1 gene in leukemia cell proliferation and apoptosis;Materials Express;2023-08-01

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