Steady-state neutrophil homeostasis is dependent on TLR4/TRIF signaling

Author:

Bugl Stefanie1,Wirths Stefan1,Radsak Markus P.2,Schild Hansjörg3,Stein Pamela3,André Maya C.4,Müller Martin R.1,Malenke Elke1,Wiesner Tina1,Märklin Melanie1,Frick Julia-Stefanie5,Handgretinger Rupert3,Rammensee Hans-Georg6,Kanz Lothar1,Kopp Hans-Georg1

Affiliation:

1. Department of Medical Oncology, Hematology, Immunology, Rheumatology and Pulmonology, Medical Center II, South West German Comprehensive Cancer Center, University Hospital of Tuebingen, Tuebingen, Germany;

2. Department of Internal Medicine III (Hematology, Oncology, Pneumology), Johannes Gutenberg University Medical Center, Mainz, Germany;

3. Institute for Immunology, Johannes Gutenberg University Medical Center, Mainz, Germany;

4. Department of Pediatric Hematology and Oncology, University Children's Hospital, Tuebingen, Germany;

5. Institute of Medical Microbiology and Hygiene, Eberhard Karls University Tuebingen, Tuebingen, Germany; and

6. Department of Immunology, Institute for Cell Biology, Eberhard Karls University Tuebingen, Tuebingen, Germany

Abstract

Key Points Steady-state and emergency granulopoiesis are both dependent on TLR signaling.

Publisher

American Society of Hematology

Subject

Cell Biology,Hematology,Immunology,Biochemistry

Reference50 articles.

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