On the molecular origins of the chronic myeloproliferative disorders: it all makes sense-Reference-Cited by-同舟云学术

On the molecular origins of the chronic myeloproliferative disorders: it all makes sense

Published:2005-06-01 Issue:11 Volume:105 Page:4187-4190
ISSN:0006-4971
Container-title:Blood
language:en
Short-container-title:

Author:

Kaushansky Kenneth¹

Affiliation:

1. From the Department of Medicine, Division of Hematology/Oncology, University of California San Diego, San Diego, CA.

Publisher

American Society of Hematology

Subject

Cell Biology,Hematology,Immunology,Biochemistry

Link

http://ashpublications.org/blood/article-pdf/105/11/4187/1708551/zh801105004187.pdf

Reference45 articles.

1. Dameshek W. Some speculations on the myeloproliferative syndromes. Blood. 1951;6: 372-375.

2. Deininger MWN, Goldman JM, Melo JV. The molecular biology of chronic myeloid leukemia. Blood. 2000;96: 3343-3356.

3. Tefferi A, Pardanani A. Clinical, genetic, and therapeutic insights into systemic mast cell disease. Curr Opin Hematol. 2004;11: 58-64.

4. Baxter EJ, Scott LM, Campbell PJ, et al. Acquired mutation of the tyrosine kinase JAK2 in human myeloproliferative diseases. Lancet. 2005;365: 1054-1061.

5. Levine RL, Wadleigh M, Cools J, et al. Activating mutation in the tyrosine kinase JAK2 in polycythemia vera, essential thrombocythemia, and myeloid metaplasia with myelofibrosis. Cancer Cell. Prepublished on March 24, 2005, as DOI 10.1016/..2005.03.023.

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