Activation of neutrophil respiratory burst by fungal particles requires phosphatidylinositol 3-phosphate binding to p40phox in humans but not in mice
Author:
Affiliation:
1. Washington University School of Medicine, St Louis, MO
2. Indiana University School of Medicine, Indianapolis, IN
Publisher
American Society of Hematology
Subject
Cell Biology,Hematology,Immunology,Biochemistry
Link
http://ashpublications.org/blood/article-pdf/120/16/3385/1498560/zh804212003385.pdf
Reference12 articles.
1. Neutrophils from p40phox−/− mice exhibit severe defects in NADPH oxidase regulation and oxidant-dependent bacterial killing.;Ellson;J Exp Med,2006
2. PtdIns3P binding to the PX domain of p40phox is a physiological signal in NADPH oxidase activation.;Ellson;EMBO J,2006
3. The phosphoinositide-binding protein p40phox activates the NADPH oxidase during FcgammaIIA receptor-induced phagocytosis.;Suh;J Exp Med,2006
4. Fc{gamma}R-stimulated activation of the NADPH oxidase: phosphoinositide-binding protein p40phox regulates NADPH oxidase activity after enzyme assembly on the phagosome.;Tian;Blood,2008
5. A new genetic subgroup of chronic granulomatous disease with autosomal recessive mutations in p40 phox and selective defects in neutrophil NADPH oxidase activity.;Matute;Blood,2009
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