HIV infection, lymphoid fibrosis, and disease
Author:
Affiliation:
1. UNIVERSITY OF CALIFORNIA, SAN FRANCISCO
Publisher
American Society of Hematology
Subject
Cell Biology,Hematology,Immunology,Biochemistry
Link
http://ashpublications.org/blood/article-pdf/120/9/1753/1362587/zh803512001753.pdf
Reference11 articles.
1. Critical role of CD4 T cells in maintaining lymphoid tissue structure for immune cell homeostasis and reconstitution.;Zeng;Blood,2012
2. Microbial translocation is a cause of systemic immune activation in chronic HIV infection.;Brenchley;Nat Med,2006
3. Valganciclovir reduces T cell activation in HIV-infected individuals with incomplete CD4+ T cell recovery on antiretroviral therapy.;Hunt;J Infect Dis,2011
4. Inflammatory and coagulation biomarkers and mortality in patients with HIV infection.;Kuller;PLoS Med,2008
5. HIV infection, inflammation, immunosenescence, and aging.;Deeks;Ann Rev Med,2011
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2. Cenicriviroc, a dual CCR2 and CCR5 antagonist leads to a reduction in plasma fibrotic biomarkers in persons living with HIV on antiretroviral therapy;HIV Research & Clinical Practice;2019-09-03
3. The Role of Transforming Growth Factor Beta-1 in the Progression of HIV/AIDS and Development of Non-AIDS-Defining Fibrotic Disorders;Frontiers in Immunology;2017-11-02
4. TGF-β inhibits IL-7-induced proliferation in memory but not naive human CD4 + T cells;Journal of Leukocyte Biology;2017-06-06
5. Circulating Biomarkers of Immune Activation Distinguish Viral Suppression from Nonsuppression in HAART-Treated Patients with Advanced HIV-1 Subtype C Infection;Mediators of Inflammation;2014
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