AID splice variants lack deaminase activity-Reference-Cited by-同舟云学术

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AID splice variants lack deaminase activity

Published:2009-02-19 Issue:8 Volume:113 Page:1862-1864
ISSN:0006-4971
Container-title:Blood
language:en
Short-container-title:

Author:

van Maldegem Febe,Scheeren Ferenc A.,Aarti Jibodh R.,Bende Richard J.,Jacobs Heinz,van Noesel Carel J. M.

Publisher

American Society of Hematology

Subject

Cell Biology,Hematology,Immunology,Biochemistry

Link

http://ashpublications.org/blood/article-pdf/113/8/1862/1488688/zh800809001862a.pdf

Reference8 articles.

1. Alternative splicing regulates activation-induced cytidine deaminase (AID): implications for suppression of AID mutagenic activity in normal and malignant B cells.;Wu;Blood,2008

2. Genome-wide somatic hypermutation.;Wang;Proc Natl Acad Sci U S A,2004

3. Pathophysiology of B-cell intrinsic immunoglobulin class switch recombination deficiencies.;Durandy;Adv Immunol,2007

4. Retroviral vectors to monitor somatic hypermutation.;Klasen;J Immunol Methods,2005

5. The APOBEC-2 crystal structure and functional implications for the deaminase AID.;Prochnow;Nature,2007

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1. A Novel AICDA Splice-Site Mutation in Two Siblings with HIGM2 Permits Somatic Hypermutation but Abrogates Mutational Targeting;Journal of Clinical Immunology;2022-03-05

2. Potential APOBEC-mediated RNA editing of the genomes of SARS-CoV-2 and other coronaviruses and its impact on their longer term evolution;Virology;2021-04

3. PDGFA-associated protein 1 protects mature B lymphocytes from stress-induced cell death and promotes antibody gene diversification;Journal of Experimental Medicine;2020-07-01

4. Activation-induced deaminase and its splice variants associate with trisomy 12 in chronic lymphocytic leukemia;Annals of Hematology;2018-10-27

5. Activation‐Induced Deaminase;eLS;2018-06-14

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