SETD2: a complex role in blood malignancy
Author:
Affiliation:
1. UNIVERSITY OF FLORIDA HEALTH CANCER CENTER
Publisher
American Society of Hematology
Subject
Cell Biology,Hematology,Immunology,Biochemistry
Link
http://ashpublications.org/blood/article-pdf/130/24/2576/1404370/blood811927.pdf
Reference10 articles.
1. SETD2 alterations impair DNA damage recognition and lead to resistance to chemotherapy in leukemia;Mar;Blood,2017
2. Intragenic DNA methylation prevents spurious transcription initiation;Neri;Nature,2017
3. The histone mark H3K36me3 regulates human DNA mismatch repair through its interaction with MutSα;Li;Cell,2013
4. Transcriptionally active chromatin recruits homologous recombination at DNA double-strand breaks;Aymard;Nat Struct Mol Biol,2014
5. Methylated α-tubulin antibodies recognize a new microtubule modification on mitotic microtubules;Park;MAbs,2016
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1. SET domain containing 2 promotes megakaryocyte polyploidization and platelet generation through methylation of α-tubulin;Journal of Thrombosis and Haemostasis;2024-06
2. Therapeutic effect of allogeneic stem cell transplantation in acute myeloid leukemia patients with epigenetic modifier gene mutations;2024-01-11
3. SETD2 Deficiency Confers Sensitivity to Dual Inhibition of DNA Methylation and PARP in Kidney Cancer;Cancer Research;2023-09-11
4. Transcriptomic Analysis Reveals JAK2/MPL-Independent Effects of Calreticulin Mutations in a C. elegans Model;Cells;2023-01-02
5. RNA mis-splicing drives viral mimicry response after DNMTi therapy in SETD2-mutant kidney cancer;Cell Reports;2023-01
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