Severe neurotoxicity following arsenic therapy for acute promyelocytic leukemia: potentiation by thiamine deficiency-Reference-Cited by-同舟云学术

Severe neurotoxicity following arsenic therapy for acute promyelocytic leukemia: potentiation by thiamine deficiency

Published:2002-05-01 Issue:9 Volume:99 Page:3481-3482
ISSN:1528-0020
Container-title:Blood
language:en
Short-container-title:

Author:

Yip Sze-Fai¹,Yeung Yiu-Ming¹,Tsui Edmond-Yik-Kong¹

Affiliation:

1. Correspondence: Sze-fai Yip, Hematology Division, Dept of Medicine, Tuen Mun Hospital, Hong Kong, China; e-mail:

Publisher

American Society of Hematology

Subject

Cell Biology,Hematology,Immunology,Biochemistry

Link

http://ashpublications.org/blood/article-pdf/99/9/3481/1682985/h80902003476d.pdf

Reference8 articles.

1. United States multicenter study of arsenic trioxide in relapsed acute promyelocytic leukemia.;Soignet;J Clin Oncol.,2001

2. Sensorimotor polyneuropathy following arsenic trioxide therapy for relapsed acute promyelocytic leukemia [abstract].;Densmore;Blood.,1999

3. Cardiac toxicity of arsenic trioxide [letter].;Singer;Blood.,2001

4. Biochemistry of some toxic agents; I, present state of knowledge of biochemical lesions induced by trivalent arsenical poisoning.;Peters;Bull John Hopkins Hosp.,1955

5. Arsenic toxicity is enzyme specific and its effects on ligation are not caused by the inhibition of DNA repair enzymes.;Hu;Mutat Res.,1998

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