Impaired Replenishment of Cortico-Striatal Synaptic Glutamate in Huntington’s Disease Mouse Model-Reference-Cited by-同舟云学术

Impaired Replenishment of Cortico-Striatal Synaptic Glutamate in Huntington’s Disease Mouse Model

Published:2020-06-05 Issue:2 Volume:9 Page:149-161
ISSN:1879-6397
Container-title:Journal of Huntington's Disease
language:
Short-container-title:JHD

Author:

Buren Caodu¹²,Tu Gaqi³⁴,Raymond Lynn A.²

Affiliation:

1. Graduate Program in Neuroscience, The University of British Columbia, Vancouver, Canada

2. Department of Psychiatry and Djavad Mowafaghian Centre for Brain Health, The University of British Columbia, Vancouver, Canada

3. School of Medicine, Zhejiang University, Hangzhou, Zhejiang, China

4. Department of Psychology, University of Toronto, Toronto, Ontario, Canada

Publisher

IOS Press

Subject

Cellular and Molecular Neuroscience,Neurology (clinical)

Reference43 articles.

1. The Huntington’s Disease Collaborative Research Group;A novel gene containing a trinucleotide repeat that is expanded and unstable on Huntington’s disease chromosomes;Cell,1993

2. Huntington’s chorea;Myrianthopoulos;J Med Genet,1966

3. Pathophysiology of Huntington’s disease: Time-dependent alterations in synaptic and receptor function;Raymond;Neuroscience,2011

4. Role of brain-derived neurotrophic factor in Huntington’s disease;Zuccato;Prog Neurobiol,2007

5. Early increase in extrasynaptic NMDA receptor signaling and expression contributes to phenotype onset in Huntington’s disease mice;Milnerwood;Neuron,2010

Cited by 2 articles. 订阅此论文施引文献订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献

1. Mechanisms of synapse‐to‐nucleus calcium signalling in striatal neurons and impairments in Huntington's disease;Journal of Neurochemistry;2024-05-21

2. Cerebrolysin Alleviating Effect on Glutamate-Mediated Neuroinflammation Via Glutamate Transporters and Oxidative Stress;Journal of Molecular Neuroscience;2022-11