Perlecan Domain V Inhibits Amyloid-β Induced Brain Endothelial Cell Toxicity and Restores Angiogenic Function
Author:
Affiliation:
1. Department of Molecular and Cellular Medicine, Texas A&M Health Science Center, College Station, TX, USA
2. Sanders-Brown Center on Aging, Department of Anatomy and Neurobiology, and Neurology, University of Kentucky, Lexington, KY, USA
Publisher
IOS Press
Subject
Psychiatry and Mental health,Geriatrics and Gerontology,Clinical Psychology,General Medicine,General Neuroscience
Cited by 20 articles. 订阅此论文施引文献 订阅此论文施引文献，注册后可以免费订阅5篇论文的施引文献，订阅后可以查看论文全部施引文献
1. Integratingin silicopredictions with an engineered tissue assay identifies Perlecan as an age-perturbed re-quiescence cue for muscle stem cells;2024-07-23
2. Perlecan: a review of its role in neurologic and musculoskeletal disease;Frontiers in Physiology;2023-05-30
3. Heparan sulfate proteoglycan in Alzheimer’s disease: aberrant expression and functions in molecular pathways related to amyloid-β metabolism;American Journal of Physiology-Cell Physiology;2023-04-01
4. Recombinant Human Perlecan DV and Its LG3 Subdomain Are Neuroprotective and Acutely Functionally Restorative in Severe Experimental Ischemic Stroke;Translational Stroke Research;2022-12-12
5. Abeta peptides disrupt the barrier integrity and glucose metabolism of human induced pluripotent stem cell-derived brain microvascular endothelial cells;NeuroToxicology;2022-03
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