Aberrant Caspase Activation in Laminin-α2-Deficient Human Myogenic Cells is Mediated by p53 and Sirtuin Activity
Author:
Affiliation:
1. Department of Neurology, Boston University School of Medicine, Boston, MA, USA
2. Whitaker Cardiovascular Institute, Boston University School of Medicine, Boston, MA, USA
Publisher
IOS Press
Subject
Neurology (clinical),Neurology
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5. Clinical and molecular study in congenital muscular dystrophy with partial laminin alpha 2 (LAMA2) deficiency;Tezak;Hum Mutat,2003
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2. A Family of Laminin α2 Chain-Deficient Mouse Mutants: Advancing the Research on LAMA2-CMD;Frontiers in Molecular Neuroscience;2020-04-21
3. Early skeletal muscle pathology and disease progress in the dy3K/dy3K mouse model of congenital muscular dystrophy with laminin α2 chain-deficiency;Scientific Reports;2019-10-04
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Current understanding and treatment of cardiac and skeletal muscle pathology in laminin-α2 chain-deficient congenital muscular dystrophy
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