Mitophagy Failure in APP and Tau Overexpression Model of Alzheimer’s Disease-Reference-Cited by-同舟云学术

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Mitophagy Failure in APP and Tau Overexpression Model of Alzheimer’s Disease

Published:2019-07-23 Issue:2 Volume:70 Page:525-540
ISSN:1387-2877
Container-title:Journal of Alzheimer's Disease
language:
Short-container-title:JAD

Author:

Martín-Maestro Patricia¹²,Gargini Ricardo¹³,García Esther¹,Simón Diana⁴,Avila Jesús¹²,García-Escudero Vega¹²

Affiliation:

1. Centro de Biología Molecular “Severo Ochoa” (UAM-CSIC), Cantoblanco, Madrid, Spain

2. Centro de Investigación Biomédica en Red de Enfermedades Neurodegenerativas (CIBERNED), Madrid, Spain

3. Neuro-oncology Unit, Instituto de Salud Carlos III-UFIEC, Majadahonda, Madrid, Spain

4. Facultad de Ciencias Experimentales, Universidad Francisco de Vitoria, Pozuelo de Alarcón, Madrid, Spain

Publisher

IOS Press

Subject

Psychiatry and Mental health,Geriatrics and Gerontology,Clinical Psychology,General Medicine,General Neuroscience

Reference53 articles.

1. Neuronal origin of a cerebral amyloid: Neurofibrillary tangles of Alzheimer’s disease contain the same protein as the amyloid of plaque cores and blood vessels;Masters;EMBO J,1985

2. Alzheimer’s disease: Genes, proteins, and therapy;Selkoe;Physiol Rev,2001

3. Evidence that the 42- and 40-amino acid forms of amyloid beta protein are generated from the beta-amyloid precursor protein by different protease activities;Citron;Proc Natl Acad Sci U S A,1996

4. Alzheimer’s disease: A dysfunction of the amyloid precursor protein(1);Neve;Brain Res,2000

5. Tau phosphorylation by GSK3 in different conditions;Avila;Int J Alzheimers Dis,2012

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