GABAB Receptor-Mediated PI3K/Akt Signaling Pathway Alleviates Oxidative Stress and Neuronal Cell Injury in a Rat Model of Alzheimer’s Disease-Reference-Cited by-同舟云学术

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GABAB Receptor-Mediated PI3K/Akt Signaling Pathway Alleviates Oxidative Stress and Neuronal Cell Injury in a Rat Model of Alzheimer’s Disease

Published:2020-08-18 Issue:4 Volume:76 Page:1513-1526
ISSN:1387-2877
Container-title:Journal of Alzheimer's Disease
language:
Short-container-title:JAD

Author:

Sun Zhiqing¹,Sun Lei²,Tu Lixiang³

Affiliation:

1. Department of Neurology, Linyi People’s Hospital, Linyi, P.R. China

2. Department of General Surgery, Linyi Hedong District People’s Hospital, Linyi, P.R. China

3. Department of Nosocomial Infection Management, Linyi People’s Hospital, Linyi, P.R. China

Publisher

IOS Press

Subject

Psychiatry and Mental health,Geriatrics and Gerontology,Clinical Psychology,General Medicine,General Neuroscience

Reference39 articles.

1. Recent progress on the role of GABAergic neurotransmission in the pathogenesis of Alzheimer’s disease;Abbas;Rev Neurosci,2016

2. Viral gene transfer of APPsalpha rescues synaptic failure in an Alzheimer’s disease mouse model;Fol;Acta Neuropathol,2016

3. Pathogenesis and disease-modifying therapy in Alzheimer’s disease: The flat line of progress;Castellani;Arch Med Res,2012

4. Update on Alzheimer’s disease therapy and prevention strategies;Graham;Annu Rev Med,2017

5. Interplay between microRNAs and oxidative stress in neurodegenerative diseases;Konovalova;Int J Mol Sci,2019

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