The Unexpected Role of Aβ1-42 Monomers in the Pathogenesis of Alzheimer’s Disease-Reference-Cited by-同舟云学术

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The Unexpected Role of Aβ1-42 Monomers in the Pathogenesis of Alzheimer’s Disease

Published:2018-03-13 Issue:3 Volume:62 Page:1241-1245
ISSN:1387-2877
Container-title:Journal of Alzheimer's Disease
language:
Short-container-title:JAD

Author:

Tamagno Elena¹²,Guglielmotto Michela¹²,Monteleone Debora¹²,Manassero Giusi¹²,Vasciaveo Valeria¹²,Tabaton Massimo³

Affiliation:

1. Department of Neuroscience, University of Torino, Torino, Italy

2. Neuroscience Institute of Cavalieri Ottolenghi Foundation (NICO), University of Torino, Torino, Italy

3. Department of Internal Medicine and Medical Specialties (DIMI), Unit of Geriatric Medicine, University of Genova, Genova, Italy

Publisher

IOS Press

Subject

Psychiatry and Mental health,Geriatrics and Gerontology,Clinical Psychology,General Medicine,General Neuroscience

Reference50 articles.

1. Alzheimer’s disease: Genes, proteins, and therapy;Selkoe;Physiol Rev,2001

2. Forecasting the global burden of Alzheimer’s disease;Brookmeyer;Alzheimers Dement,2007

3. Abeta and tau form soluble complexes that may promote self aggregation of both into the insoluble forms observed in Alzheimer’s disease;Guo;Proc Natl Acad Sci USA,2006

4. Induction of tau pathology by intracerebral infusion of amyloid-beta -containing brain extract and by amyloid-beta deposition in APP x Tau transgenic mice;Bolmont;Am J Pathol,2007

5. Bioinorganic chemistry of Alzheimer’s disease;Kepp;Chem Rev,2012

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