Impact of the Maraviroc-Resistant Mutation M434I in the C4 Region of HIV-1 gp120 on Sensitivity to Antibody-Mediated Neutralization
Author:
Affiliation:
1. Center for AIDS Research, Kumamoto University
2. AIDS Research Center, National Institute of Infectious Diseases
3. AIDS Clinical Center, National Center for Global Health and Medicine
Publisher
Editorial Committee of Japanese Journal of Infectious Diseases, National Institute of Infectious Dis
Subject
Infectious Diseases,Microbiology (medical),General Medicine
Link
https://www.jstage.jst.go.jp/article/yoken/69/3/69_JJID.2015.310/_pdf
Reference55 articles.
1. 1. Berger EA, Murphy PM, Farber JM. Chemokine receptors as HIV-1 coreceptors: roles in viral entry, tropism, and disease. Annu Rev Immunol. 1999;17:657-700.
2. 2. Dorr P, Westby M, Dobbs S, et al. Maraviroc (UK-427, 857), a potent, orally bioavailable, and selective small-molecule inhibitor of chemokine receptor CCR5 with broad-spectrum anti-human immunodeficiency virus type 1 activity. Antimicrob Agents Chemother. 2005;49:4721-32.
3. 3. Asin-Milan O, Chamberland A, Wei Y, et al. Mutations in variable domains of the HIV-1 envelope gene can have a significant impact on maraviroc and vicriviroc resistance. AIDS Res Ther. 2013;10:15.
4. 4. Berro R, Sanders RW, Lu M, et al. Two HIV-1 variants resistant to small molecule CCR5 inhibitors differ in how they use CCR5 for entry. PLoS Pathog. 2009;5:e1000548.
5. 5. Delobel P, Raymond S, Mavigner M, et al. Shift in phenotypic susceptibility suggests a competition mechanism in a case of acquired resistance to maraviroc. AIDS. 2010;24:1382-4.
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