Influence of ADAMTS13 deficiency on venous thrombosis in mice
Author:
Funder
Ministry of Health
Publisher
Georg Thieme Verlag KG
Subject
Hematology
Link
http://www.thieme-connect.de/products/ejournals/pdf/10.1160/TH14-08-0656.pdf
Reference20 articles.
1. von Willebrand factor–mediated platelet adhesion is critical for deep vein thrombosis in mouse models
2. Monocytes, neutrophils, and platelets cooperate to initiate and propagate venous thrombosis in mice in vivo
3. Neutrophil Extracellular Trap (NET) Impact on Deep Vein Thrombosis
4. Thrombogenesis with continuous blood flow in the inferior vena cava
5. Extracellular DNA traps promote thrombosis
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1. Impact of the von Willebrand factor-ADAMTS-13 axis on the risk of future venous thromboembolism;Journal of Thrombosis and Haemostasis;2023-05
2. ADAMTS13 Inhibits H2O2-induced Human Venous Endothelial Cell Injury to Attenuate Deep-Vein Thrombosis by Blocking the p38/ERK Signaling Pathway;Chinese Journal of Physiology;2023
3. von Willebrand Factor Is a Critical Mediator of Deep Vein Thrombosis in a Mouse Model of Diet-Induced Obesity;Arteriosclerosis, Thrombosis, and Vascular Biology;2020-12
4. Plasminogen Tochigi mice show reduced fibrinolytic activity but exhibit phenotypes similar to wild-type mice under experimental thrombotic conditions;Japanese Journal of Thrombosis and Hemostasis;2018
5. Plasminogen Tochigi mice exhibit phenotypes similar to wild-type mice under experimental thrombotic conditions;PLOS ONE;2017-07-07
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