Relationship between Serum Copper, Ceruloplasmin, and Non–Ceruloplasmin-Bound Copper in Routine Clinical Practice

Author:

Twomey Patrick J1,Viljoen Adie2,House Ivan M3,Reynolds Timothy M4,Wierzbicki Anthony S5

Affiliation:

1. Department of Clinical Biochemistry, The Ipswich Hospital, Ipswich, United Kingdom

2. Department of Chemical Pathology, Addenbrooke’s Hospital, Cambridge, United Kingdom

3. The Medical Toxicology Unit Laboratory, Guy’s & St Thomas’ Hospital Trust, London, United Kingdom

4. Department of Chemical Pathology, Queen’s Hospital, Burton-on-Trent, United Kingdom

5. Department of Chemical Pathology, St Thomas’ Hospital, London, United Kingdom

Publisher

Oxford University Press (OUP)

Subject

Biochemistry (medical),Clinical Biochemistry

Reference12 articles.

1. Gollan JL, Gollan TJ. Wilson disease in 1998: genetic, diagnostic and therapeutic aspects. J Hepatol1998;28:28-36.

2. On-Line Mendelian Inheritance in Man. Wilson disease. OMIM 277900. http://www.ncbi.nlm.nih.gov/omim (accessed June 26, 2004)..

3. Steindl P, Ferenci P, Dienes HP, Grimm G, Pabinger I, Madl C, et al. Wilson’s disease in patients presenting with liver disease: a diagnostic challenge. Gastroenterology1997;113:212-218.

4. Gibbs K, Walshe JM. A study of caeruloplasmin concentrations found in 75 patients with Wilson’s disease, their kinships and various control groups. Q J Med1979;48:447-463.

5. Milne DB. Trace elements. Burtis CA Ashwood ER eds. Tietz textbook of clinical chemistry, 3rd ed1999:1041-1044 WB Saunders Philadelphia. .

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