Regulation of the Phosphatidylinositide 3-Kinase Pathway by the Lipid Phosphatase PTEN
Author:
Affiliation:
1. The Campbell Family Institute for Breast Cancer Research
2. Department of Medical Biophysics, University of Toronto, and
3. Ontario Cancer Institute, University Health Network, Toronto, Ontario, Canada
Publisher
Oxford University Press (OUP)
Subject
Biochemistry (medical),Clinical Biochemistry
Link
http://academic.oup.com/clinchem/article-pdf/62/6/884/32644579/clinchem0884.pdf
Reference5 articles.
1. Identification of a candidate tumour suppressor gene, MMAC1, at chromosome 10q23.3 that is mutated in multiple advanced cancers;Steck;Nat Genet,1997
2. The tumor suppressor, PTEN/MMAC1, dephosphorylates the lipid second messenger, phosphatidylinositol 3,4,5-trisphosphate;Maehama;J Biol Chem,1998
3. High cancer susceptibility and embryonic lethality associated with mutation of the PTEN tumor suppressor gene in mice;Suzuki;Curr Biol,1998
4. Nuclear PTEN controls DNA repair and sensitivity to genotoxic stress;Bassi;Science,2013
5. PTEN;Worby;Annu Rev Biochem,2014
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3. Retracted: Targeted Regulation of miR-26a on PTEN to Affect Proliferation and Apoptosis of Prostate Cancer Cells;Cancer Biotherapy and Radiopharmaceuticals;2019-09-01
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