Knockdown of UCA1 inhibits viability and glycolysis by suppressing PKM2 expression through the mTOR pathway in non-small cell lung cancer cells-Reference-Cited by-同舟云学术

Knockdown of UCA1 inhibits viability and glycolysis by suppressing PKM2 expression through the mTOR pathway in non-small cell lung cancer cells

Published:2018 Issue:19 Volume:8 Page:10610-10619
ISSN:2046-2069
Container-title:RSC Advances
language:en
Short-container-title:RSC Adv.

Author:

Wang Xuguang¹²³⁴,Fa Xian-En⁵⁶⁷⁴^ORCID

Affiliation:

1. Department of Thoracic Surgery

2. The First Affiliated Hospital of Zhengzhou University

3. Zhengzhou 450052

4. P. R. China

5. Department of Cardiothoracic Surgery

6. The Second Affiliated Hospital of Zhengzhou University

7. Zhengzhou 450014

Abstract

LncRNA urothelial carcinoma associated 1 (UCA1) was reported to be upregulated in non-small cell lung cancer (NSCLC) tissues and contributed to NSCLC progression.

Publisher

Royal Society of Chemistry (RSC)

Subject

General Chemical Engineering,General Chemistry

Link

http://pubs.rsc.org/en/content/articlepdf/2018/RA/C8RA00860D

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